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T1 - Anatomy of prostaglandin signals

The most important job of Series 3 prostaglandins is to prevent AA from being released by cells, thus preventing the production of bad Series 2 prostaglandins.

Prostaglandins have a variety of biochemical and physiological effects, including hormonal effects.

So, if you've been reading the chemical literature much in the last 5 years or so, you've probably noticed the ongoing explosion of papers on the topic of Photoredox chemistry. It seems that organic chemists in the field have been borrowing increasingly bizarre transition metal complexes from the electroluminescence, photophysical and materials literature for some time now, and keeping track of them all is a real pain. I mean, how many people can draw from memory the structure of that Bernhard Ir(4',6'-dF-5-CF3-ppy)2(4,4'-dtbbpy)PF6 catalyst from the latest MacMillan group paper? Don't look at me like that—it's commercially available from Aldrich.[1] And you can remember its oxidation potential, right? Versus the ? In the Ir(iii)* excited state and as Ir(iv) after reductive quenching?

A prostaglandin synthesis - [PDF Document]

T1 - Increased prostaglandin synthesis by low calcium-regulated keratinocytes

These results suggest that the 40% ethanol extract of mangosteen has potent inhibitory activities of both histamine release and prostaglandin E2 synthesis.

Its efficacy appears to be related to induction of glutathione S-transferase enzymes, inhibition of prostaglandin E(2) (PGE(2)) production, or suppression of oxidative DNA adduct (M(1)G) formation.

prostaglandin synthesis - Pctrucos Pesquisador


We previously showed pomegranate seed oil and fermented juice polyphenols to retard oxidation and prostaglandin synthesis, to inhibit breast cancer cell proliferation and invasion, and to promote breast cancer cell apoptosis.

N2 - The effect of calcium concentration on the in vitro prostaglandin production by murine keratinocytes was studied using radioimmunoassay. Keratinocytes grown in low-calcium medium (0.02 mm) maintained intracellular calcium levels adequate for arachidonic acid metabolism and actually showed increased prostaglandin production. Baseline, unstimulated PGE2 production was 4.5 times higher in cells growing in low- compared to normal-calcium (1.2 mm) medium (p = 0.001). PGF2α production was increased 2.5 times in the low-calcium cells (p = 0.002). The calcium ionophore A23187 and 12-O-tetradecanoyl-phorbol-13-acetate (TPA) exhibited differing calcium requirements for activation of the arachidonic acid pathway. A23187 ionophore stimulated prostaglandin synthesis only in cells growing in normal-calcium medium while TPA stimulated prostaglandin production by both low- and normal-calcium cells. Paradoxically, short-term exposure of low calcium-grown cells to normal-calcium medium abolished the TPA effect. These results suggested that calcium can control arachidonic acid metabolism at a number of regulatory points.

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Stereo-controlled synthesis of dl-prostaglandins …

The effect of calcium concentration on the in vitro prostaglandin production by murine keratinocytes was studied using radioimmunoassay. Keratinocytes grown in low-calcium medium (0.02 mm) maintained intracellular calcium levels adequate for arachidonic acid metabolism and actually showed increased prostaglandin production. Baseline, unstimulated PGE2 production was 4.5 times higher in cells growing in low- compared to normal-calcium (1.2 mm) medium (p = 0.001). PGF production was increased 2.5 times in the low-calcium cells (p = 0.002). The calcium ionophore A23187 and 12-O-tetradecanoyl-phorbol-13-acetate (TPA) exhibited differing calcium requirements for activation of the arachidonic acid pathway. A23187 ionophore stimulated prostaglandin synthesis only in cells growing in normal-calcium medium while TPA stimulated prostaglandin production by both low- and normal-calcium cells. Paradoxically, short-term exposure of low calcium-grown cells to normal-calcium medium abolished the TPA effect. These results suggested that calcium can control arachidonic acid metabolism at a number of regulatory points.

A prostaglandin synthesis - ResearchGate

AB - The effect of calcium concentration on the in vitro prostaglandin production by murine keratinocytes was studied using radioimmunoassay. Keratinocytes grown in low-calcium medium (0.02 mm) maintained intracellular calcium levels adequate for arachidonic acid metabolism and actually showed increased prostaglandin production. Baseline, unstimulated PGE2 production was 4.5 times higher in cells growing in low- compared to normal-calcium (1.2 mm) medium (p = 0.001). PGF2α production was increased 2.5 times in the low-calcium cells (p = 0.002). The calcium ionophore A23187 and 12-O-tetradecanoyl-phorbol-13-acetate (TPA) exhibited differing calcium requirements for activation of the arachidonic acid pathway. A23187 ionophore stimulated prostaglandin synthesis only in cells growing in normal-calcium medium while TPA stimulated prostaglandin production by both low- and normal-calcium cells. Paradoxically, short-term exposure of low calcium-grown cells to normal-calcium medium abolished the TPA effect. These results suggested that calcium can control arachidonic acid metabolism at a number of regulatory points.

23/12/2017 · A prostaglandin synt..

For patients with peripheral arterial occlusive disease stage III or IV not eligible for arterial reconstruction, Prostaglandin E1 (PGE1) therapy not only has significant beneficial effects over placebo on ulcer healing and pain relief but also increases the rate of patients surviving with both legs after 6-months follow-up.
Prostaglandin E1 may be beneficial for some patients with erectile dysfunction.

| The total synthesis of dl-prostaglandin F1 alpha is reported

Nonsteroidal anti-inflammatory medications (NSAIDs) and selective cyclooxygenase-2 (COX-2) inhibitors reduce Prostaglandin E2 production to diminish the inflammation seen in these diseases, but have toxicities that may include both gastrointestinal bleeding and prothrombotic tendencies.

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